Lecture 16
      
                              Models of Psychopathology
      
      
      Lecture Outline
      
      I.  Introduction
      II. Medical Model
           A. Criticisms
      III.Biopsychosocial Model
           A. Characteristics
                1. Hierarchical Organization
                2. Reverberating System
           B. Implications
      IV. Discussion: A Deficit Model?
           A. Blaming the Victim
           B. Meta-messages and Self-fulfilling Prophecies
      V.  Difference Model
      VI.  Conclusion
      
      
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      I.   Introduction
      
           We have looked at numerous factors that are associated with the
      etiology of various psychological disorders.  We've examined biology,
      physiology, genetics, learning, social factors, cognitions,
      communication, emotions, and so on.  The question is, how do we put all
      these various factors together?  Are certain factors more important than
      others in determining the etiology of psychological disorders?  Is there
      one factor that is primary; all the others being secondary outcomes of
      the one primary factor?  So far we've really only looked at these
      various etiological factors in isolation - a catalog of hypotheses if
      you will.  We need to see if there is a way to organize these factors
      into an overarching model of psychopathology.  Without such a unifying
      model, a comprehensive understanding of psychopathology will continue to
      elude us.  
           Today we will examine some attempts at providing this overarching
      framework.  We will look at various models of psychopathology.  Each
      model attempts to answer (at least some of) the questions we have posed. 
      This discussion is a starting place:  it is not an exhaustive list of
      models, nor is it the last time we will confront these issues.
       
      II. The Medical Model
      
           The dominant model today (at least within psychiatry) is the
      medical model of psychopathology (Carpenter, 1987; Engel, 1980).  The
      basic assumption is that psychological disorders are diseases.  The
      nature of onset, distribution of cases, development and course,
      treatment response, and associated features seen in psychological
      disorders are seen to be parallel to what occurs in physical diseases
      (Carpenter, 1987).  This model assumes diseases of any sort to be fully
      understood in terms of abnormal biological variables (Engel, 1977). 
      Thus, a "psychological" disorder can be explained in terms of (and
      actually is) a disorder of underlying physical mechanisms (e.g.:
      biochemical and physiological processes).  Of the etiological factors
      that we have examined, the biological realm is primary.  To understand
      psychopathology, we need not look beyond the biological level.
           This approach embraces reductionism:  a philosophical view that
      complex phenomena (such as thoughts, behaviors, emotions) can be
      completely understood and explained in terms of a more basic level. 
      That is, in this case, thoughts, behaviors and emotions can be "reduced
      to" the more basic level of biological processes.  A thought is a
      neurological event in the brain.  Psychopathology is a biological
      phenomenon.  
           Implications:  Research and treatment will focus on searching for
      and altering biological variables. 
      
      A. Criticisms
      
           This model has been criticized (e.g: Carpenter, 1987; Engel,
      1980) as being insufficient for truly understanding
      psychopathology.  "The crippling flaw of the (medical) model is
      that it...can make provision neither for the person as a whole nor
      for data of a psychological or social nature" (Engel, 1980).  
           Biology simply can't account for psychological disorders.  A
      model of psychological phenomena must be based on other levels of
      data, levels that involve psychological processes; for example:
      cognitive and social levels.  At the very least, to truly
      understand a psychological disorder, we need to integrate knowledge
      from these various levels with the biological level.  We need to
      recognize that each level has its own strengths, but also its own
      limitations.  Biological levels do pretty good at providing
      explanations of "form", that is, it answers "how" questions:  how a
      particular disease process occurs, what its mechanisms are, etc. 
      Biological explanations do not, however, provide explanations of
      the "function" of the disorder.  That is, biology does not address
      the "why" questions:  why did this disorder occur, what is its
      meaning, purpose, or function?  Both sets of questions are
      important in understanding a phenomenon.  Both approaches need to
      be assimilated.
      
      An everyday example  
      
      This will illustrate the necessity of integrating various
      levels of analysis to fully understand a phenomenon.  Let's
      look at blushing (Carpenter, 1987).  We could reduce a blush
      to the biological level: vascular changes in the body.  But
      this certainly is not the whole story.  At best, we know the
      physiological mechanism of how the blush occurs.  But we also
      need to look at why it occurs.  We need to recognize the role
      that psychological variables play: a person blushes when they
      experience shame, for example; and we need to look at
      sociological variables: the blush occurs in response to shame
      when the person is in a public setting.  No single level
      (biology, psychology or social) is sufficient in providing an
      explanation of blushing.
           
      Another Example
      
      In 1982, a group of Danish doctors reported that five
      previously healthy and skilled men who were severely tortured
      six years before the doctors had examined them had experienced
      various forms of brain atrophy (Stover & Nelson, 1985).  For
      example:  enlarged cerebral ventricular structures (Carpenter,
      1987).  Such a phenomenon can not be explained at just one
      level.  A comprehensive explanation requires that we look at
      physiological mechanisms that induce structural changes in
      response to psychological processes, such as pain, associated
      with torture.  In other words, we must integrate biological,
      psychological and social factors.
      
      III. The Biopsychosocial Model
      
           An alternative model, the biopsychosocial model, attempts to
      recognize the shortcomings of the medical model (Engel, 1977, 1980).  A
      basic assumption remains: the "disease model" is the essential frame of
      reference (Carpenter, 1987).  However, the biopsychosocial model, in
      contrast to the medical model, conceptualizes disease as a multilevel
      phenomenon.  It rejects the reductionistic bias of the medical model. 
      To embrace reductionism means important data will be excluded.  
           For example, let us look at Schizophrenia (Carpenter, 1987).  To
      fully understand Schizophrenia, we must understand the nature of the
      disorder at multiple levels.  It is the interaction between these levels
      that account for the manifestations of Schizophrenia.  It is not at all
      a priori evident which level is primary.  For example:  Social changes
      may lead to brain changes, but so too will brain changes lead to social
      changes.  Is one always the cause of the other?  Which one?  Or are
      there different independent causes for changes of the brain and changes
      at the social level?  Or do they both have a common causative factor?
           The biopsychosocial level identifies numerous levels that may be
      relevant in understanding psychopathology, including social,
      psychological, biological, and physical variables (Carpenter, 1987). 
      Figure 16-1 provides a more detailed list of the various levels.
      
      A. Characteristics
      
      There are two basic characteristics of the biopsychosocial model:
      
      1. Hierarchical Organization:  The levels listed in Figure 16-
      1 can be organized in a hierarchical continuum.  The more
      complex, larger units can be seen as superordinate to the less
      complex, smaller units.  In this way, each level of the
      hierarchy represents an organized system with distinct
      properties and characteristics.  And each level will thus
      requires its own methods of study unique to that level (Engel,
      1980).
      
      2.  Reverberating System:  The hierarchically arranged levels
      can also be seen as part of an overarching system, where
      activity at one level influences other levels - the activity
      at one level reverberates to other levels (Carpenter, 1987). 
      This can be seen in Figure 16-2 where each level is at the
      same time a component of higher levels.  Thus, every level is
      at the very same time "both a whole and a part".  "Nothing
      exists in isolation".  For example, we can look at the level
      of red blood cells, study them and explain them.  But in so
      doing we are also implying the existence of the larger systems
      without which the red blood cell has no existence (Engel,
      1980).  
      
      B. Implications
      
           The comprehensive study of psychological disorders will
      require research in many areas.  Hopes for finding the single,
      unitary hypothesis for a disorder will be futile.
           This does not negate the importance of doing narrow, specific
      experiments.  Such studies are necessary for exploring specific
      associations and processes and for examining predicted
      relationships.  Such experiments can provide a foundation upon
      which to build.  The problem arises when investigators stop at that
      level and do not integrate their knowledge with other levels of
      investigation.
           Psychopathology is very complex.  So too, then, will be the
      methods of study and the theories we build.  It has been argued my
      various scientists that the medical model presents an overly
      simplistic picture of psychopathology:  The boundaries between
      health and disease are not at all clear:  they are wrapped up in
      cultural, social and psychological considerations as well as
      biological considerations (eg: Engel, 1977).
      
      IV. Discussion: A Deficit Model?
      
           The biopsychosocial model has much to offer.  It sees the person as
      a whole; it recognizes the complexity inherent in psychological
      disorders.  Nevertheless, the biopsychosocial model is open to
      criticisms as well.
           One of the strongest criticisms of the biopsychosocial model has
      been that it is a deficit model of psychopathology.  That is, it sees as
      the core factor in psychological "illness" a problem with the person. 
      There is something about the person that is amiss, whether it is "under
      his/her skin" or in his/her relation to the world.  So, to understand a
      disorder, we search for abnormalities in subjects' biology,
      relationships, and the like.  On the face of it, such an approach seems
      logical.  Of course there is something wrong with the person.  
      
      A. Blaming the Victim
      
           However, such a deficit orientation has been challenged as
      being actually a case of blaming the victim (Ryan, 1976).  Critics
      claim that people suffering from "mental illness" are actually the
      victims of an environment that is hostile to them.  For example: 
      The economy is in terrible shape, the person is a woman and a
      minority in a prejudiced community, and she is unemployed with 3
      children to take care of.  She becomes depressed, and eventually
      suicidal.  The biopsychosocial model certainly will take into
      account all these factors, but (and here's the point) it will still
      place the locus of the problem within the woman.  She is suffering
      from a psychological disorder.  This, claim some, is blaming the
      victim, which only serves to hurt the person we label as ill.
      
      B. Meta-messages and Self-Fulfilling Prophecies
      
           Critics of this deficit-model orientation claim that it
      communicates certain things to people who are diagnosed with
      psychological disorders and to people in general.  Such an
      orientation communicates messages of weakness.  It is telling the
      mental health patient that he/she has deficits and defects, that
      other people without them are healthy, and the more he/she can be
      like them the better he/she will be.  The message is one of
      inferiority.  Not only does the person come to believe this (after
      all, experts are saying so), so too does the community in general. 
      The person so labelled, as well as others, will overgeneralize from
      the label - the label ends up doing more harm than what ever
      problems the person might originally have actually had. 
           The outcome, critics continue, is that mental illness becomes
      associated with stigma.  To be diagnosed is to be labelled.  That
      label has a lot of stigma attached to it.  People misunderstand it. 
      We can see such stigma in action:  Presidential candidate Mike
      Dukakis had all sorts of problems when it was suspected that he
      might have been treated for depression.  
           The stigma communicated to patients produces a self-fulfilling
      prophecy:  the message of inferiority is internalized by the
      patients, so they come to believe in their inferiority and act
      accordingly.  There is a long history of research that stresses the
      importance of others' expectations, attitudes, and appraisals about
      an individual for that individual's identity formation and self-
      esteem (e.g.:  Cooley, 1964;  Mead, 1962; Rosenberg, 1965).  
      
      V. A Difference Model
      
           What critics claim is that the shift from the medical model to the
      biopsychosocial model really made no fundamental change in orientation. 
      The underlying nature of these two models is the same:  the deficit
      model.  The victim blaming, meta-messages and self-fulfilling prophecies
      that the two imply are just the same.  They both are models that
      conceptualize the "patient" as defective or deficient in some way.
           So what do the critics offer as an alternative?  One alternative
      orientation is to use a difference model (Rappaport, 1977).  The basic
      assumptions are changed.  The question is no longer what is wrong with
      the person, but what are the strengths of this person and how can they
      be used.  The goal changes from rehabilitating the person, to finding a
      setting into which the person can fit and use his/her abilities, and
      where he/she can develop new abilities.  These critics are calling for
      "a psychology of strengths rather than weaknesses" (Rappaport, 1977,
      p.125).  
           From this perspective, we would approach the schizophrenic person,
      for example, in a very different manner than we did in the other models. 
      The focus now (Rappaport, 1977): 
      
      emphasize an "individual" rather than a "patient" status
      
      treat person as responsible human being (eg: expected to  
      participate in their own self-care:  work, recreational,
      social activities)
      
      rather than providing "treatment" (so person can fit back 
      into society), restructure society so their are more     
      opportunities and resources available for the person and
      what skills s/he does have
      
      it's as much other people's responsibility to change as   
      it is the "patient's" - cannot merely remove "patients"   
      form society and place them in hospitals
      
      all people ("patients" and "nonpatients") should live in  
      mutually supportive ways, in the community if possible
      
      
      VI. Conclusion
      
           We began by examining two of the major approaches to
      psychopathology research and theory:  the medical model and the
      biopsychosocial model.  The latter has clear advantages over the former:
      a more sophisticated and comprehensive understanding of psychological
      disorder is possible.  Such a biopsychosocial is really a call for
      interdisciplinary cooperation in the study of psychopathology, a
      cooperation that has not historically been a reality.  Nevertheless, as
      we have seen, there are some criticisms that can be leveled at both the
      medical and biopsychosocial models.  If they do indeed embody
      assumptions of weakness and deficits, then there may be undesirable
      implications for how we conceptualize and treat people with (so called?)
      psychological disorders.  Some scientists thus advocate a difference
      model for understanding psychopathology.  
           Once again, we seem to have run into: "just exactly what do we mean
      by Psychopathology?"  I am certainly not advocating one of these models
      as The Best or The Correct model...But I am saying this:  Again, I urge
      you not to be lulled into the belief that everything is clear-cut and
      simple.  Stay sharp!  Look for assumptions!  And question them!  No
      science advances with blind acceptance of what's, "of course", common-
      sense.  By comparing and contrasting these various models, we have
      learned about some of their strengths and weaknesses; their uses and
      potential misuses.  This is always a useful exercise!